The Role of Gut Microbiota in Gastrointestinal Cancers: From Pathogenesis to Therapeutic Perspectives 2.0

A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Cancer Biology and Oncology".

Deadline for manuscript submissions: closed (31 March 2024) | Viewed by 449

Special Issue Editor


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Guest Editor
Department of Gastroenterological Endoscopy, Tokyo Medical University, Tokyo, Japan
Interests: gastric cancer; intestinal metaplasia; gastritis; tumor immunity; helicobacter pylori; gastric microbiota; endoscopy; cancer surveillance; cancer chemoprevention; colorectal cancer; artificial intelligence
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Special Issue Information

Dear Colleagues,

This Special Issue will focus on the role of gut microbiota in gastrointestinal cancers, from pathogenesis to therapeutic perspectives, and old and new tools used to evaluate carcinogenesis in vitro and in vivo.

We emphasize the relevance of driver microbiota that accelerate inflammation and carcinogenesis, such as Fusobacterium spp. and others. This Issue includes reviews, opinions, and short articles from experts in the field.

Submissions will cover all types of microbiota, including bacteria, fungi, viruses, and bacteriophages that influence carcinogenesis and molecular pathways/factors playing critical roles in persistent inflammation, various mouse models, and valuable cell lines, primary organoids, and comparative studies with other tumor entities.

We also expand on our issue between metabolites derived from these microbiomes and carcinogenesis.

Review manuscript submissions focusing on innovative therapeutic targets and potential clinical trial candidates, including the molecular mechanisms and the interplay among different signaling pathways, are welcome.

We hope this Special Issue will help improve our understanding of the role of gut microbiota in gastrointestinal cancers.

Dr. Ryota Niikura
Guest Editor

Manuscript Submission Information

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Keywords

  • gastric cancer
  • colon cancer
  • esophageal cancer
  • microbiota
  • metaplasia
  • mouse model
  • organoid
  • Helicobacter
  • Fusobacterium spp.
  • eradication
  • signaling pathways
  • cell crosstalk
  • genetics
  • immunological milieu

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