Macrophages during Inflammation
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cellular Immunology".
Deadline for manuscript submissions: closed (1 March 2021) | Viewed by 8674
Special Issue Editors
Interests: pathogen/host interaction during candidiasis; innate immune response; trained immunity; PRRs; hematopoietic stem and progenitor cells; myeloid cells; macrophages
Interests: hematopoietic stem and progenitor cells; myelopoiesis; myeloid cells; monocytes; macrophages; dendritic cells; infection; Candida albicans; innate immune memory; trained immunity
Special Issue Information
Dear Colleagues,
Macrophages are critical mediators of tissue homeostasis. They are generally considered to be one of the first lines of defense against pathogens, as they employ a range of strategies to phagocytose and kill pathogens and play important roles in orchestrating the protective inflammatory response through pattern recognition receptors. In addition, macrophages play an important role in removing cellular debris and repairing tissues. They are strategically positioned throughout the body’s tissues, either established prenatally (embryonic origin) and maintained through adulthood or derived from tissue-infiltrating monocytes (hematopoietic origin).
For this Special Issue entitled "Macrophages during Inflammation", we welcome the submission of original research and review articles covering a broad range of topics, including: macrophage-pathogen interactions in infectious diseases: detection, phagocytosis, and inflammatory responses; molecular mechanisms for inflammatory signaling in macrophages; plasticity of macrophage function; tolerance and trained immunity in macrophages; immunomodulation of macrophage function; and influence of origin on macrophage functional phenotype. Furthermore, macrophages are important to the development of several human diseases. Therefore, this Special Issue may also include topics related to pulmonary, cardiovascular, or metabolic inflammatory diseases, neurodegeneration, aging, or cancer.
We look forward to your contribution.
Dr. María Luisa Gil
Dr. Alberto Yáñez
Guest Editors
Manuscript Submission Information
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Keywords
- infection
- phagocytosis
- signaling
- inflammatory mediators
- origins
- tolerance
- trained immunity
- immune-response-mediated diseases
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