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Correction

Correction: Zhang et al. Deficiency of S100A9 Alleviates Sepsis-Induced Acute Liver Injury through Regulating AKT-AMPK-Dependent Mitochondrial Energy Metabolism. Int. J. Mol. Sci. 2023, 24, 2112

Beijing Key Laboratory of Cardiopulmonary Cerebral Resuscitation, Department of Emergency Medicine, Beijing Chaoyang Hospital, Capital Medical University, Beijing 100020, China
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Authors to whom correspondence should be addressed.
Int. J. Mol. Sci. 2025, 26(3), 935; https://doi.org/10.3390/ijms26030935
Submission received: 9 January 2025 / Accepted: 9 January 2025 / Published: 23 January 2025
(This article belongs to the Section Molecular Endocrinology and Metabolism)
In the original publication [1], there was a mistake in Figure 4D as published. The image of the H&E staining in the CLP-KO+CC group in Figure 4D was shown incorrectly during the manuscript preparation. The correct version of Figure 4D is included below. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Zhang, Y.; Wu, F.; Teng, F.; Guo, S.; Li, H. Deficiency of S100A9 Alleviates Sepsis-Induced Acute Liver Injury through Regulating AKT-AMPK-Dependent Mitochondrial Energy Metabolism. Int. J. Mol. Sci. 2023, 24, 2112. [Google Scholar] [CrossRef] [PubMed]
Figure 4. Inhibition of AMPK reverses S100A9-KO-mediated protection of CLP-induced liver dysfunction. (A) Diagram of AMPK inhibitor Compound C (CC). (B) Schematic diagram of WT or S100A9-knockout (KO) mice treated with CC and CLP operation for 24 h. (C) The levels of serum ALT and AST in each group (n = 6). (D) H&E staining of liver sections (left), and quantification of the histological score (right, n = 6). (E) TUNEL (red) and DAPI (blue) staining of liver sections (left), and quantification of TUNEL+ nuclei (right, n = 6). (F) Immunohistochemical staining of liver sections with anti-CD68 antibody (left), and quantification of the CD68+ area (right, n = 6). (G) DHE staining of liver sections to measure superoxide production (left), and quantification of fluorescence intensity (right, n = 6). Data are shown as mean ± SEM, and n represents the number of mice in each group.
Figure 4. Inhibition of AMPK reverses S100A9-KO-mediated protection of CLP-induced liver dysfunction. (A) Diagram of AMPK inhibitor Compound C (CC). (B) Schematic diagram of WT or S100A9-knockout (KO) mice treated with CC and CLP operation for 24 h. (C) The levels of serum ALT and AST in each group (n = 6). (D) H&E staining of liver sections (left), and quantification of the histological score (right, n = 6). (E) TUNEL (red) and DAPI (blue) staining of liver sections (left), and quantification of TUNEL+ nuclei (right, n = 6). (F) Immunohistochemical staining of liver sections with anti-CD68 antibody (left), and quantification of the CD68+ area (right, n = 6). (G) DHE staining of liver sections to measure superoxide production (left), and quantification of fluorescence intensity (right, n = 6). Data are shown as mean ± SEM, and n represents the number of mice in each group.
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MDPI and ACS Style

Zhang, Y.; Wu, F.; Teng, F.; Guo, S.; Li, H. Correction: Zhang et al. Deficiency of S100A9 Alleviates Sepsis-Induced Acute Liver Injury through Regulating AKT-AMPK-Dependent Mitochondrial Energy Metabolism. Int. J. Mol. Sci. 2023, 24, 2112. Int. J. Mol. Sci. 2025, 26, 935. https://doi.org/10.3390/ijms26030935

AMA Style

Zhang Y, Wu F, Teng F, Guo S, Li H. Correction: Zhang et al. Deficiency of S100A9 Alleviates Sepsis-Induced Acute Liver Injury through Regulating AKT-AMPK-Dependent Mitochondrial Energy Metabolism. Int. J. Mol. Sci. 2023, 24, 2112. International Journal of Molecular Sciences. 2025; 26(3):935. https://doi.org/10.3390/ijms26030935

Chicago/Turabian Style

Zhang, Yanting, Feng Wu, Fei Teng, Shubin Guo, and Huihua Li. 2025. "Correction: Zhang et al. Deficiency of S100A9 Alleviates Sepsis-Induced Acute Liver Injury through Regulating AKT-AMPK-Dependent Mitochondrial Energy Metabolism. Int. J. Mol. Sci. 2023, 24, 2112" International Journal of Molecular Sciences 26, no. 3: 935. https://doi.org/10.3390/ijms26030935

APA Style

Zhang, Y., Wu, F., Teng, F., Guo, S., & Li, H. (2025). Correction: Zhang et al. Deficiency of S100A9 Alleviates Sepsis-Induced Acute Liver Injury through Regulating AKT-AMPK-Dependent Mitochondrial Energy Metabolism. Int. J. Mol. Sci. 2023, 24, 2112. International Journal of Molecular Sciences, 26(3), 935. https://doi.org/10.3390/ijms26030935

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