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Zanni G, Hsiao C-T, Fu S-J, Tang C-Y, Capuano A, Bosco L, Graziola F, Bellacchio E, Servidei S, Primiano G, et al. Novel KCND3 Variant Underlying Nonprogressive Congenital Ataxia or SCA19/22 Disrupt K_V4.3 Protein Expression and K+ Currents with Variable Effects on Channel Properties. International Journal of Molecular Sciences. 2021; 22(9):4986. https://doi.org/10.3390/ijms22094986

Zanni, Ginevra, Cheng-Tsung Hsiao, Ssu-Ju Fu, Chih-Yung Tang, Alessandro Capuano, Luca Bosco, Federica Graziola, Emanuele Bellacchio, Serenella Servidei, Guido Primiano, and et al. 2021. "Novel KCND3 Variant Underlying Nonprogressive Congenital Ataxia or SCA19/22 Disrupt K_V4.3 Protein Expression and K+ Currents with Variable Effects on Channel Properties" International Journal of Molecular Sciences 22, no. 9: 4986. https://doi.org/10.3390/ijms22094986

Zanni, G., Hsiao, C. -T., Fu, S. -J., Tang, C. -Y., Capuano, A., Bosco, L., Graziola, F., Bellacchio, E., Servidei, S., Primiano, G., Soong, B. -W., & Jeng, C. -J. (2021). Novel KCND3 Variant Underlying Nonprogressive Congenital Ataxia or SCA19/22 Disrupt K_V4.3 Protein Expression and K+ Currents with Variable Effects on Channel Properties. International Journal of Molecular Sciences, 22(9), 4986. https://doi.org/10.3390/ijms22094986