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Correction

Correction: Wang et al. Nicotinic Acetylcholine Receptor Alpha6 Contributes to Antiviral Immunity via IMD Pathway in Drosophila melanogaster. Viruses 2024, 16, 562

Department of Entomology, China Agricultural University, Beijing 100193, China
*
Authors to whom correspondence should be addressed.
Viruses 2024, 16(5), 742; https://doi.org/10.3390/v16050742
Submission received: 23 April 2024 / Accepted: 24 April 2024 / Published: 8 May 2024
(This article belongs to the Special Issue Molecular Virus-Insect Interactions)

Error in Table

In the original publication [1], there was a mistake in “Table 1. Fly stocks” as published. The fly stock BL67048 rather than TB00129 was used in our study. But it was written incorrectly in the writing process of this article.
The corrected “Table 1. Fly stocks” appears below.
Table 1. Fly stocks.
Table 1. Fly stocks.
Gene TypeStock IDSource
w*; P{UAS-3xFLAG.dCas9.VPR} attP40; P{tubP-GAL4} LL7/T(2;3)TSTL14, SM5:TM6B, Tb1BL67048Bloomington Drosophila Stock Center, Bloomington, IN, USA
if/cyo; daughterlessGal4TB00153Tsinghua Stock Center, Beijing, China
Dα6KODRZ17-CG4128-1CCT related Drosophila line generated by Dr. Yi Rao’s Lab at Peking University, Beijing, China [10]
w1118V60000Vienna Drosophila Resource Center, Vienna, Austria
AttB-RNAiV52342Vienna Drosophila Resource Center, Vienna, Austria
y1sc*v1sev21; P{TOE.GS01969}attP40BL80510Bloomington Drosophila Stock Center, Bloomington, IN, USA
y1sc*v1sev21; P{GS00089} attP40BL67539Bloomington Drosophila Stock Center, Bloomington, IN, USA
w1118; RelE20esBL9457Bloomington Drosophila Stock Center, Bloomington, IN, USA
if/cyo; MKRS/TM6B, Tb-Obtained from Genetics Department, University of Cambridge, Cambridge, United Kingdom
-22a (DMelSV susceptible strain)Obtained from Genetics Department, University of Cambridge, Cambridge, United Kingdom

Text Correction

There was an error in the original publication [1]. BL67048 and y1sc*v1sev21; P {TOE.GS01969} attP40 can be crossed to obtain a Dα6-overexpression line, while TB00129 could not obtain it.
A correction has been made to “2. Materials and Methods “, “2.1. Flystocks”, “Paragraph 3”:
BL67048 was crossed with BL80510 to obtain the Dα6-overexpression strain P {TOE.GS01969} attP40/ P{UAS-3xFLAG.dCas9.VPR} attP40; P{tubP-GAL4} LL7. The control line P{GS00089} attP40/ P{UAS-3xFLAG.dCas9.VPR} attP40; P{tubP-GAL4} LL7 was generated by crossing BL67048 and BL67539. Daughterless-Gal4 was crossed with V52342 to obtain knock-down line daughterless-Gal4; AttB-RNAi, and the progeny daughterless-Gal4; a cross between daughterless-Gal4 and V60000 was used as the control.
The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Wang, Z.; Lin, X.; Shi, W.; Cao, C. Nicotinic Acetylcholine Receptor Alpha6 Contributes to Antiviral Immunity via IMD Pathway in Drosophila melanogaster. Viruses 2024, 16, 562. [Google Scholar] [CrossRef] [PubMed]
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MDPI and ACS Style

Wang, Z.; Lin, X.; Shi, W.; Cao, C. Correction: Wang et al. Nicotinic Acetylcholine Receptor Alpha6 Contributes to Antiviral Immunity via IMD Pathway in Drosophila melanogaster. Viruses 2024, 16, 562. Viruses 2024, 16, 742. https://doi.org/10.3390/v16050742

AMA Style

Wang Z, Lin X, Shi W, Cao C. Correction: Wang et al. Nicotinic Acetylcholine Receptor Alpha6 Contributes to Antiviral Immunity via IMD Pathway in Drosophila melanogaster. Viruses 2024, 16, 562. Viruses. 2024; 16(5):742. https://doi.org/10.3390/v16050742

Chicago/Turabian Style

Wang, Zhiying, Xiaoju Lin, Wangpeng Shi, and Chuan Cao. 2024. "Correction: Wang et al. Nicotinic Acetylcholine Receptor Alpha6 Contributes to Antiviral Immunity via IMD Pathway in Drosophila melanogaster. Viruses 2024, 16, 562" Viruses 16, no. 5: 742. https://doi.org/10.3390/v16050742

APA Style

Wang, Z., Lin, X., Shi, W., & Cao, C. (2024). Correction: Wang et al. Nicotinic Acetylcholine Receptor Alpha6 Contributes to Antiviral Immunity via IMD Pathway in Drosophila melanogaster. Viruses 2024, 16, 562. Viruses, 16(5), 742. https://doi.org/10.3390/v16050742

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