Nucleic Acid Sensing in Respiratory Diseases—Series 2
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cell Nuclei: Function, Transport and Receptors".
Deadline for manuscript submissions: closed (15 May 2024) | Viewed by 205
Special Issue Editors
Interests: injury-induced inflammation; chronic progressive respiratory diseases; viral superinfection; innate immune danger sensing; cGAS/STING; inflammasome activation; IL-1 family; Th2; Th17; altered microbiota; metabolites; tissue homeostasis; inflammatory responses; translational research in mouse models of human disease
Special Issues, Collections and Topics in MDPI journals
Interests: pulmonar immunology; immunopharmacology of pulmonary diseases; chemokines; cell recruitment; pulmonary inflammation; pulmonary fibrosis; asthma and infection by influenza virus
Special Issue Information
Dear Colleagues,
This is the second edition of Nucleic Acid Sensing in Respiratory Diseases, which previously published 8 papers.
The recognition of host cell or pathogen-derived nucleic acids by pattern recognition receptors (PRRs) is essential to elicit host innate immune responses in numerous contexts, such as inflammatory diseases as well as auto-inflammatory or tumor settings. We intend to review the recent development in this field, including your recent discoveries in this thematic issue of Cells.
Basic aspects of cell stress-induced nucleic acid sensing and recent developments in the mechanisms of nucleic acid-mediated cell activation are welcome. Specifically, we invite studies on inflammatory and infectious diseases in general, including respiratory ones. Translational considerations and potential novel therapeutic targets will be of great interest.
Prof. Dr. Bernhard Ryffel
Dr. Remo Russo
Guest Editors
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Keywords
- cGAS
- STING
- inflammasome
- TLR
- infection
- cell stress
- lung inflammation
- inhibitors
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Related Special Issue
- Nucleic Acid Sensing in Respiratory Diseases in Cells (8 articles)
