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10th Anniversary of JCDD—'Pediatric Cardiology and Congenital Heart Disease'...
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10th Anniversary of JCDD—'Pediatric Cardiology and Congenital Heart Disease' Section

A special issue of Journal of Cardiovascular Development and Disease (ISSN 2308-3425). This special issue belongs to the section "Pediatric Cardiology and Congenital Heart Disease".

Deadline for manuscript submissions: 20 July 2025 | Viewed by 4433

Special Issue Editor

Prof. Dr. Natasja de Groot

E-Mail Website1 Website2
Guest Editor
Department of Cardiology, Erasmus Medical Center, 3015 GD Rotterdam, The Netherlands
Interests: cardiac mapping; mechanism of cardiac arrhythmias; signal recording- and processing technologies
Special Issues, Collections and Topics in MDPI journals

Special Issue Information

Dear Colleagues,

The cardiac surgery of congenital heart disease has evolved significantly over the past decades. With advances in medical technology, diagnosis and treatment are now possible at earlier stages, improving not only the chances of survival but also outcomes for patients. There has been an increased focus on a holistic treatment approach by dedicated cardiac care teams, including surgeons, pediatric and adult congenital cardiologists, cardiologist electrophysiologists, cardiac anesthesiologists, cardiac pathologists and cardiac intensive care specialists. This approach has resulted in considerable improvements in the quality of life and outcomes for patients.

There has also been significant progress in research on the molecular and genetic basis of congenital heart disease and associated complications such as cardiac arrhythmias, which has led to a better understanding of the underlying mechanisms. However, despite all the tremendous progress in the diagnosis and treatment of congenital heart disease, the main challenge at present is to effectively treat the late postoperative sequala cardiac arrhythmias and heart failure.

In this Special Issue, we discuss the evolution in the clinical care of patients with congenital disease, discuss the pathophysiology of congenital heart disease, associated conditions and future developments and highlight the knowledge gaps.

Prof. Dr. Natasja de Groot
Guest Editor

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Journal of Cardiovascular Development and Disease is an international peer-reviewed open access monthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • cardiac surgery
  • procedural outcome
  • postoperative course
  • postoperative sequela
  • cardiac arrhythmias
  • heart failure
  • genetic defects
  • survival rates

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Published Papers (2 papers)

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Research

13 pages, 1134 KiB  
Article
Prophylactic Pulmonary Artery Banding in Pediatric Dilated Cardiomyopathy: An Additional Therapeutic Option
by Elena Panaioli, Diala Khraiche, Margaux Pontailler, Flavie Ader, Olivier Raisky, Regis Gaudin and Damien Bonnet
J. Cardiovasc. Dev. Dis. 2024, 11(3), 79; https://doi.org/10.3390/jcdd11030079 - 27 Feb 2024
Viewed by 2097
Abstract
Dilated cardiomyopathy (DCM) is the most common childhood cardiomyopathy and is associated with considerable early mortality. Heart transplantation is often the only viable life-saving option. Pulmonary artery banding (PAB) has been recently proposed as a bridge or alternative to transplantation for DCM. In [...] Read more.
Dilated cardiomyopathy (DCM) is the most common childhood cardiomyopathy and is associated with considerable early mortality. Heart transplantation is often the only viable life-saving option. Pulmonary artery banding (PAB) has been recently proposed as a bridge or alternative to transplantation for DCM. In our cohort, PAB was selectively addressed to heritable DCM or DCM with congenital left ventricle aneurysm (CLVA). This study aimed to describe the clinical evolution and left ventricle reverse remodeling (LVRR) over time (6 months and 1 year after surgery). Ten patients with severe DCM received PAB between 2016 and 2021 and underwent clinical and postoperative echocardiography follow-ups. The median age at PAB was <1 year. The in-hospital mortality was zero. Two patients died two months after PAB of end-stage heart failure. The modified Ross class was improved in the eight survivors with DCM and remained stable in the two patients with CLVA. We observed a positive LVRR (LV end-diastolic diameter Z-score: 8.4 ± 3.7 vs. 2.8 ± 3; p < 0.05; LV ejection fraction: 23.8 ± 5.8 to 44.5 ± 13.1 (p < 0.05)). PAB might be useful as part of the armamentarium available in infants and toddlers with severe DCM not sufficiently responding to medical treatment with limited probability of spontaneous recovery. Full article
(This article belongs to the Special Issue 10th Anniversary of JCDD—'Pediatric Cardiology and Congenital Heart Disease' Section)
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8 pages, 806 KiB  
Article
Electrocardiographic Changes with Age in Japanese Patients with Noonan Syndrome
by Yasuhiro Ichikawa, Hiroyuki Kuroda, Takeshi Ikegawa, Shun Kawai, Shin Ono, Ki-Sung Kim, Sadamitsu Yanagi, Kenji Kurosawa, Yoko Aoki, Mari Iwamoto and Hideaki Ueda
J. Cardiovasc. Dev. Dis. 2024, 11(1), 10; https://doi.org/10.3390/jcdd11010010 - 28 Dec 2023
Viewed by 1738
Abstract
Little information is available on age-related electrocardiographic changes in patients with Noonan syndrome. This single-center study evaluated the electrocardiograms of patients with Noonan syndrome. We divided the patients (n = 112; electrocardiograms, 256) into four groups according to age: G1 (1 month–1 [...] Read more.
Little information is available on age-related electrocardiographic changes in patients with Noonan syndrome. This single-center study evaluated the electrocardiograms of patients with Noonan syndrome. We divided the patients (n = 112; electrocardiograms, 256) into four groups according to age: G1 (1 month–1 year), G2 (1–6 years), G3 (6–12 years), and G4 (>12 years). Typical Noonan syndrome-related electrocardiographic features such as left-axis deviation, abnormal Q wave, wide QRS complex, and small R wave in precordial leads were detected. A high percentage of QRS axis abnormalities was found in all groups. Significant differences in right-axis deviation (RAD) were noted among the groups: 56.5% of G1 patients showed RAD compared with 33.3% of G2, 21.1% of G3, and 19.2% of G4 patients. The small R was also significantly different among the groups: 32.6% of G1 patients showed a small R wave compared with 14.9% of G2, 8.5% of G3, and 15.4% of G4 patients. Of the 53 patients with Noonan syndrome aged 1 month to 2 years, 18 had T-positive V1 with a higher prevalence of pulmonary stenosis and cardiac interventions. QRS axis abnormalities, small R in V6, and T-positive V1 could help diagnose Noonan syndrome in infants or young children. Full article
(This article belongs to the Special Issue 10th Anniversary of JCDD—'Pediatric Cardiology and Congenital Heart Disease' Section)
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