Article Versions Notes

Matzer I, Voglhuber J, Kiessling M, Djalinac N, Trummer-Herbst V, Mabotuwana N, Rech L, Holzer M, Sossalla S, Rainer PP, et al. β-Adrenergic Receptor Stimulation Maintains NCX-CaMKII Axis and Prevents Overactivation of IL6R-Signaling in Cardiomyocytes upon Increased Workload. Biomedicines. 2022; 10(7):1648. https://doi.org/10.3390/biomedicines10071648

Matzer, Ingrid, Julia Voglhuber, Mara Kiessling, Nataša Djalinac, Viktoria Trummer-Herbst, Nishani Mabotuwana, Lavinia Rech, Michael Holzer, Samuel Sossalla, Peter P. Rainer, and et al. 2022. "β-Adrenergic Receptor Stimulation Maintains NCX-CaMKII Axis and Prevents Overactivation of IL6R-Signaling in Cardiomyocytes upon Increased Workload" Biomedicines 10, no. 7: 1648. https://doi.org/10.3390/biomedicines10071648

Matzer, I., Voglhuber, J., Kiessling, M., Djalinac, N., Trummer-Herbst, V., Mabotuwana, N., Rech, L., Holzer, M., Sossalla, S., Rainer, P. P., Zirlik, A., & Ljubojevic-Holzer, S. (2022). β-Adrenergic Receptor Stimulation Maintains NCX-CaMKII Axis and Prevents Overactivation of IL6R-Signaling in Cardiomyocytes upon Increased Workload. Biomedicines, 10(7), 1648. https://doi.org/10.3390/biomedicines10071648