Molecular Biology of Retinal Ganglion Cells-Series II

Dear Colleagues,

Following the successful publication of the first Special Issue of “Molecular Biology of Retinal Ganglion Cells”, we are very pleased to launched the new edition, which is dedicated to highlighting the recent development in this field.

Retinal ganglion cells (RGCs) are the output neurons of the vertebrate retina and, in addition to integrating information and passing it to target neurons in retinorecipient brain centers, these also perform a considerable amount of computation in order to encode signals into action potential trains. This mechanism requires the coordinated expression, activation, modulation, deactivation, and disintegration of molecules that partake in RGC signaling, homeostatic processes, the maintenance of cellular integrity, and the adaptation of cells to changing conditions. While many of these molecular constituents are generally expressed by most neurons in the central nervous system, the expression of others is characteristic of RGCs, and together they form the RGC molecular fingerprint. Besides pan-RGC molecules, the over 20 morphological/functional RGC subtypes may also express unique molecules or common molecules in unique combinations, thereby providing subtype specific molecular fingerprints that can be utilized for both identification and functional considerations. Such cellular fingerprints, however, are not ‘static’ but can change during development, at times of pathological insults, or even through normal functioning. This is the Series-2, a continuation of our Special Issue that aims to bring together both original research papers and review articles addressing the ever-growing field of the molecular biology of retinal ganglion cells. Our goal is to encourage scientists in this field to contribute to this Special Issue with their related work. Suggested topics include (but are not limited to): subtype-specific RGC marker molecules, molecules of RGC signaling, and disease-induced molecular changes.

Prof. Dr. Béla Völgyi
Guest Editor

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• ganglion cells
• molecular phenotyping
• intracellular cascade
• signal processing
• development
• pathology
• adaptation
• circadian rhythm