Prematurity-Related Morbidities in Neonates: From Pathophysiology to Novel Therapeutic Approaches
A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Cell Biology and Pathology".
Deadline for manuscript submissions: closed (31 May 2023) | Viewed by 15779
Special Issue Editor
Interests: bronchopulmonary dysplasia; patent ductus arteriosus; preterm infants
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Significant advances in perinatology and neonatology in the last decade have resulted in the increased survival of extremely premature infants, leading to a lower threshold for their resuscitation. Because these extremely premature infants who can survive easily develop life-threatening diseases, such as bronchopulmonary dysplasia (BPD), patent ductus arteriosus (PDA), intraventricular hemorrhage (IVH), and retinopathy of prematurity (ROP), the rates of prematurity-related morbidities either have not been changing or have been increasing. Furthermore, because neonates who are affected by these illnesses have been getting more and more immature over the past couple of decades, the pathophysiology of these morbidities has been changing with time. Therefore, under such circumstances, prematurity-related morbidities have been poorly understood in terms of pathophysiology and lack effective treatments.
We invite investigators to contribute original research as well as review articles addressing recent advances in aspects regarding the pathophysiology of prematurity-related morbidities and in novel therapies and their effects, not only on short-term but also on long-term outcomes in children born extremely prematurely. At the same time, research regarding the understanding of the molecular mechanisms based on pharmacological treatments is welcome. Original, high-quality contributions that are not yet published or that are not currently under review by other journals or peer-reviewed conferences are sought.
Dr. Fumihiko Namba
Guest Editor
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