Molecular Advances in Gut Microbiota and Its Interaction with Immune System
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Immunology".
Deadline for manuscript submissions: 23 March 2025 | Viewed by 1067
Special Issue Editor
Special Issue Information
Dear Colleagues,
The composition of the gut microbiome plays an important role in the overall health of the host, including the maintenance of immunological homeostasis. Numerous studies have revealed that powerful molecular interactions between various taxa of the gut microbiome and its counteractive host immune system components take place. The purpose of this Special Issue is to facilitate the presentation of such findings with emphasis on the formulation of novel hypotheses that might lead to interventional therapies and/or treatments.
As the research from the past two decades indicates, critical areas include, but are not limited to:
- Gut dysbiosis vs. chronic intestinal/systemic inflammation.
- Diet-induced gut dysbiosis vs. pathogenesis of mucosa-associated/systemic disease.
- Gut microbiome and cancer.
- Gut dysbiosis vs. autoimmunity (Inflammatory Bowel Disease, Celiac Disease, etc.).
- Gut dysbiosis vs. development of neuropathological disorders.
- Identification of gut microbiota-derived immunomodulatory metabolites including their impact on intestinal epithelium, i.e., gut permeability.
- Elucidation of novel host epithelial cell derived micro RNAs and their interactions with specific species of host gut microbiome.
- Identification of new therapeutical interventions capable of modulating and reversing gut dysbiosis.
In summary, in this Special Issue, we welcome original research articles, as well as review/opinion articles with a focus on molecular interactions between the gut microbiome and host immune system.
Dr. Karol Sestak
Guest Editor
Manuscript Submission Information
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Keywords
- gut microbiota
- dysbiosis
- gut permeability
- intestinal inflammation
- autoimmunity
- IBD
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