Interplay between Influenza Virus and Host Factors
A special issue of Viruses (ISSN 1999-4915). This special issue belongs to the section "Animal Viruses".
Deadline for manuscript submissions: 31 December 2024 | Viewed by 1338
Special Issue Editor
Interests: molecular virology; influenza virus; viral pathogenesis; virus–host interactions; innate antiviral response; post-translational protein modifications; protein trafficking; acetylation; HDACs; HATs
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Influenza virus has posed a challenge to global public health for centuries and will continue to do so for the foreseeable future due to its genetic makeup, diversity, and broad host range. Influenza virus is an obligate intracellular pathogen and interacts with numerous host factors to complete its life cycle in the host. These interactions are both exploitative and antagonistic and contribute to the host adaptation and disease severity of influenza virus. Hence, the host factors involved in the life cycle of influenza virus may be divided into two categories—proviral and antiviral—even if some host factors might play a dual role. Influenza virus utilizes all three main parts—plasma membrane, cytoplasm, and nucleus—of a host cell to complete its life cycle. Influenza virus continues to be the subject of extensive research, and new findings on the interplay between influenza virus and host factors continue to be demonstrated. A detailed understanding of the interplay between influenza virus and host factors is key in elucidating the mechanisms of viral pathogenesis and design-targeted antiviral strategies. This Special Issue of Viruses invites articles reporting the latest research developments in this exciting area of influenza virus research.
Dr. Matloob Husain
Guest Editor
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Keywords
- influenza virus
- host factors
- proviral host factors
- antiviral host factors
- host adaptation
- virus entry
- virus assembly
- virus release
- innate antiviral response
- interferon-stimulated genes
- post-translational protein modifications
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