Redox Control of Cardiac and Skeletal Muscle Function II
A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "Health Outcomes of Antioxidants and Oxidative Stress".
Deadline for manuscript submissions: closed (30 November 2022) | Viewed by 18380
Special Issue Editor
Interests: cancer and chemotherapy; cardiac and skeletal muscle; mitochondrial dysfunction; redox balance and exercise
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
The supraphysiological production of reactive oxygen species (ROS) in cardiac and skeletal muscle is a hallmark of numerous conditions associated with muscle contractile dysfunction, including but not limited to disease, inactivity, and aging. Equilibrium within the redox system is necessary to control the activation of signaling pathways, which are essential for the maintenance of cardiac and skeletal muscle function. Conversely, oxidation may lead to changes in protein activities that promote beneficial adaptations in gene expression. Thus, a more comprehensive understanding of the mechanisms by which altered redox regulation can modulate signaling pathways, which, in turn, regulate muscle contractile protein function, is needed to develop therapeutic interventions to prevent cardiac and skeletal muscle dysfunction resulting from a diverse array of conditions.
This second edition Special Issue welcomes manuscripts that could fill the gap between mechanistic understanding of redox signaling and utilization of antioxidative therapies as they relate to disease and wasting conditions. This issue includes but is not limited to the following themes:
- Redox control of cardiac and skeletal muscle atrophy and/or dysfunction;
- Use of antioxidative drugs to counteract cardiac and skeletal muscle atrophy and/or dysfunction;
- Emerging antioxidant therapies;
- Novel mechanisms regulating redox signaling;
- Exercise and its effect on redox balance in cardiac and skeletal muscle.
Dr. Ashley J. Smuder
Guest Editor
Manuscript Submission Information
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